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출처: 이북나라 전자책 『KSBMB』 2쪽 · 20쪽 · 38쪽 · 57쪽 · 77쪽 · 95쪽. 원문에 없는 주제·저자·발행일은 넣지 않았습니다.

KSBMB

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[B. Cell Biology/Stem Cell] B-1 Pathological Mechanism of a Constitutively Active Form of Stromal Interaction Molecule 1 (STIM1) in Skeletal Muscle Ji Hee Park1,2, Seung Yeon Jeong1,2, Jun Hee Choi1,2, Eun Hui Lee1,2* 1Department of Physiology, College of Medicine, The Catholic University of Korea, Seoul 06591, Korea, 2Department of Biomedicine & Health Sciences, Graduate School, The Catholic University of Korea, Seoul 06591, Korea STIM1 is the main protein that, along with Orai1, mediates SOCE in skeletal muscle.

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[B. Cell Biology/Stem Cell] B-10 Fe65 promotes the degradation of Jagged1 by forming a trimeric complex with Jagged1 and the E3 ligase Neurl1 Eun-Hye Jo1, Mi-Yeon Kim1, So-I Noh1, Hyung-Ju Lee1, Hee-Sae Park1* 1School of Biological Sciences and Technology, Chonnam National University, Gwangju 61186, Korea Fe65 is a highly conserved adaptor protein that interacts with several binding partners. Fe65 binds proteins to mediate various cellular processes. But the interacting partner and the regulatory mechanisms controlled by Fe65 are largely unknown.

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[B. Cell Biology/Stem Cell] B-19 Differentiation of Various Gastric Cancer Cell Lines using Glycome Signatures by LC-MS/MS Jihyeon Nam1,2, Nari Seo1,2, J Eugene Lee3, Hyun Joo An1,2* 1Graduate School of Analytical Science and Technology, Chungnam National University, Daejeon 34134, Korea, 2Asia-Pacific Glycomics Reference Site, Chungnam National University, Daejeon 34134, Korea, 3Center for Bioanalysis, Korea Research Institute of Standards and Science, Daejeon 34113, Korea Glycosylation, which is highly sensitive to the external environment, has been considered as a window reflecting…

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Discovering Therapeutic agonists to stimulate TLR3-mediated signaling in regeneration Yukyung Park1, Jiyeon Moon1, Wooyoung Lee1, Minji Kim1, Garyung Kwon1, Mina Ma1, Dongwon Kim1* 1 Bio-chemical Engineering, Dongseo University, Busan 47011, Korea Introduction Results Toll-Like-Receptor 3 (TLR3) is a family of Toll-Like-Receptor (TLR) A 8 which recognize pathogen associated molecular patterns (PAMPs) from microbes and its associated Innate immune response. A Recent 6 study demonstrated that damaged tissue induced noncoding dsRNA 4 which activates TLR3 and promotes retinoic acid in the body.

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Functional Validation of RKIP knockout effect on Cancer Cell Signaling Md Abu Rayhan , Eun-Young Lee , Mohammad Faysal Al Mazid , Minjeong Kim , Hye Jin You 1,2* ** Presenting Author : Md Abu Rayhan 1 *Corresponding Author 1 1Department of Cancer Biomedical Science, National Cancer Center (NCC)-Graduate School of Cancer Science and Policy (GCSP), Gyeonggi 10408, Korea 2 Tumor Microenvironment Branch, Division of Cancer Biology, National Cancer Center, Gyeonggi 10408, Korea. Abstract Abstract : Colon cancer, one of common cancers is relatively well characterized.

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본문 95쪽

Isolation of lysosomal activator regulating lysosomal damages and cell death Heemin Park , Shin-Hyeon Ryu , Seo-hyun Kim , and Yong-Keun Jung *,1 1 Apoptosis and Molecular Disease Laboratory; School of biological science, Seoul National University, Seoul 08826, Korea Abstract Lysosomes contribute to maintaining cellular homeostasis via degradation and recycling of subcellular substances called autophagy, along with a protective action against foreign materials, such as pathogens, and cellular protein aggregates.

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