이 책의 분량과 형식은 어떻게 되나요?

전체 8쪽 분량의 온라인 플립북입니다. 별도 프로그램을 설치하지 않고 브라우저에서 바로 넘겨 볼 수 있습니다.

이 소개의 내용은 어디에서 가져왔나요?

출처: 이북나라 전자책 『P. Molecular medicine and imaging』 1쪽 · 2쪽 · 4쪽 · 5쪽 · 7쪽 · 8쪽. 원문에 없는 주제·저자·발행일은 넣지 않았습니다.

P. Molecular medicine and imaging

본문 1쪽

Cell-Permeable Frataxin (CP-FXN) Ameliorate The Symptoms Of Friedreich’s Ataxia Through Regulation Of Cellular Iron Metabolism Joonno Lee, Sookeun Yeon, Wonheum Nah, Jeongheon Choi, Sujung Lee, Sunwoong Kim,Youngsil Choi and Daewoong Jo FXN Team, Cellivery R&D Institute, Cellivery Therapeutics, Inc., Seoul 03929, Korea BACKGROUND AIM Friedreich’s ataxia (FRDA) is monogenic autosomal recessive disease causing To ameliorate FRDA symptoms, cell-permeable FXN neurodegeneration and cardiomyopathy.

출처: 이 전자책 1쪽

본문 2쪽

[P. Molecular medicine and imaging-1] Mitochondrial Targeting Of Frataxin Rescues Abnormal Iron Accumulation By Activating Iron-Sulfur Cluster Biosynthesis Joonno Lee1, Sookeun Yeon1, Keumyoung Kang1, Jeongheon Choi1, Wonheum Na1, Sujug Lee1, Youngsil Choi1, Daewoong Jo1 ̇* 1Cellivery R&D Institute, Cellivery Therapeutics, Seoul 03929, Korea Friedreich’s ataxia (FRDA) is monogenic autosomal recessive disease causing neurodegeneration and cardiomyopathy.

출처: 이 전자책 2쪽

본문 4쪽

[P. Molecular medicine and imaging-2] Frataxin Replenishment Ameliorates Heart Dysfunction Through Recovery Of Iron Metabolism In Frataxin Deficient Mice Wonheum Nah1, Joonno Lee1, Keumyoung Kang1, Jeongheon Choi1, SooJung Lee1, Youngsil Choi1, Daewoong Jo1 1Cellivery R&D Institute, Cellivery Therapeutics, Seoul 03929, Korea Friedreich’s ataxia (FRDA) is a progressive cardiac and neurodegenerative disease with early life mortality resulted from cardiomyopathy. FRDA is caused by a deficiency of frataxin (FXN) which plays a role in regulating iron metabolism.

출처: 이 전자책 4쪽

본문 5쪽

Paeoniflorin, a Major Compound of P. lactoflora, Increases Endometrial Receptivity by Upregulating the Expression of LIF Hye-Rin Park 1,2,† , Hee-Jung Choi 1,† , Mi-Ju Park , Dong-Ryeol Ryu , Ki-Tae Ha 1,2,* Korean Medical Research Center for Healthy Aging, Pusan National University and Department of Korean Medical science, School of Korean Medicine, Pusan National University, Yangsan, Gyeongsangnam-do, Republic of Korea Department of Molecular Cell Biology, Sungkyunkwan University School of Medicine, Suwon, Republic of Korea These authors contributed equally in this study.

출처: 이 전자책 5쪽

본문 7쪽

Protein Nanobio Lab sat UNIST Target-switchable Intracellular Bacterial Toxin Delivery Systems with improved therapeutic efficacy against target tumor Seong Guk Park, Bongseo Choi, Yoonji Bae, Sebyung Kang Department of Biological Sciences, School of Life Sciences, Ulsan National Institute of Science and Technology (UNIST), Ulsan, 44919, Korea Abstract Scheme Targeted cancer therapies have been extensively developed to selectively suppress tumor growth as well as to avoid harming healthy tissues.

출처: 이 전자책 7쪽

본문 8쪽

[P. Molecular medicine and imaging-5] Target-switchable Intracellular Bacterial Toxin Delivery Systems with improved therapeutic efficacy against target tumor Seong Guk Park1 ̇#, Bongseo Choi1 ̇2 ̇#, Yoonji Bae1, Yu Geon Lee1, Soo Ah Park3, Young Chan Chae1 ̇*, Sebyung Kang1 ̇* 1Department of Biological Sciences, School of Life Sciences, Ulsan National Institute of Science and Technology (UNIST), Ulsan 44919, Korea, 2Department of Radiology, Feinberg School of Medicine, Northwestern University, Chicago 60611, United States, 3In Vivo Research Center, Ulsan National Institute of Science and…

출처: 이 전자책 8쪽